Preliminary reports indicate conolidine may perhaps inhibit particular ion channels, lowering neuronal excitability and restricting soreness signals. This mechanism is especially appropriate in neuropathic ache, where irregular signaling causes persistent soreness. Furthermore, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to soreness perception. Co... https://walterj369uqm9.blogoscience.com/profile